Insulin, is it Safe?


After its development in the 1920s, manmade insulin was deemed a “miracle cure.”  Indeed, it has prevented suffering and countless lives. Before the development of synthetic insulin, diabetes patients were given a bleak prognosis and suffered terribly from disease complications such as blindness, gout, and gangrene. Insulin has extended the lifespan of childhood diabetics from months to decades. Today, many diabetic children live normal, productive lives.

The problem with insulin is that it is prescribed universally, and not just to those with true insulin deficiencies. Giving this hormone to a person with already sufficient level does nothing to correct the underlying problem. Furthermore, its use can be counterproductive. This is because insulin stimulates the development of antagonists in the body that counteract its blood sugar-lowering effects.

When a diabetic receives insulin, and his/her blood sugar begins to fall, the body immediately responds with an output of growth hormones and epinephrine. These hormones keep blood-sugar levels elevated. The result of aggressive insulin therapy, then, is a rebound effect. 

Constant fluctuations in blood sugar can lead to a wide range of disorders. Clinical studies show that diabetics treated aggressively with insulin have a 40% greater incidence of eye problems than those treated moderately. Nevertheless, aggressive insulin treatment is the method choice in mainstream medicine for diabetics with eye problems.  


Another problem with insulin treatment is that it may contribute to inner arterial wall damage and lead to cardiovascular problems. In fact, the incidence of heart attacks and stroke is five to eight times greater among diabetics than in general population. In fact, 75% of all diabetic mortalities are due to heart disease brought on by hardening of major arteries.


Other complications of insulin are the result of damage to microvascular vessels, particularly those leading to the eyes, kidneys, and peripheral nerves.  As these arteries became thick and brittle, it is more difficult for blood to pass through them, and they become less functional. In the eyes, sudden surges of blood sugar created repeated stresses that cause these vessels to hemorrhage and break down. After several hemorrhages, blindness is a likely outcome. This is the second most cause of blindness in older people, following glaucoma.


In the kidneys, similar events can cause renal insufficiency, so that nitrogen wastes can no longer be eliminated from the body efficiently. Interference with blood circulation in both large and small vessels is responsible for the high incidence of neuritis and gangrene, which begin with sensations of tingling and ends with loss of feeling. A frequent outcome of this problem is amputation.